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"The stations you provide are strikingly similar to those I came across during my medical school finals (some even verbatim!), and I have tried many other exam platforms. I'm truly grateful for your priceless support throughout my final couple of years at medical school!"
Raza Q π¬π§
"It has absolutely everything for medical school, so many histories with detailed differential diagnoses, how to approach emergencies, commonly prescribed drugs..every kind go examination youβll ever need in osces"
John R π¬π§
"Thank you SO MUCH for the amazing educational resource. Iβve tried lots of platforms and books with mock OSCE stations and yours is by far and away the best Iβve tried"
Ed M π³πΏ
"Get this right away. So helpful for OSCEs but also general clinical learning and understanding. Wish I had brought it sooner"
Emma W π¬π§
"Without a doubt, your platform outshines all other OSCE resources currently available. In all honesty, I can confidently attribute my success in securing a distinction in my finals to OSCEstop."
Harish K π¬π§
"OSCEstop distinguishes itself from many other platform banks by offering a wealth of questions that mimic the demanding and complex aspects of our finals. This platform played a crucial role in ensuring I was ready for the level of difficulty that awaited me in my final exams."
Extradural Haemorrhage (EDH) is a type of traumatic brain injury where blood accumulates between the inner surface of the skull and the outer layer of the dura mater, the outermost meningeal layer.
Epidemiology:
Commonly seen in young adults and children.
Often associated with motor vehicle accidents, falls, or sports injuries.
Pathophysiology:
Typically caused by a tear in the middle meningeal artery, secondary to a skull fracture.
The accumulation of blood in the epidural space leads to increased intracranial pressure, compression of brain tissues, and potential herniation.
Clinical Features:
Initial loss of consciousness, followed by a lucid interval, then progressive deterioration in consciousness.
Headache, nausea, vomiting.
Focal neurological deficits such as hemiparesis.
Signs of increased intracranial pressure, including altered mental status and pupillary dilation (usually ipsilateral to the hematoma).
Diagnosis:
CT scan of the head is the diagnostic modality of choice, typically showing a biconvex (lentiform) hematoma.
MRI can be used in certain cases for further evaluation.
Management:
Neurosurgical intervention is often required, usually involving craniotomy to evacuate the hematoma.
Medical management of increased intracranial pressure.
Monitoring in an intensive care setting, especially for signs of herniation and neurological deterioration.
Prognosis:
The outcome depends on the size of the hematoma, the extent of brain injury, and the rapidity of intervention.
Early diagnosis and prompt surgical treatment generally result in a good prognosis.